What the claims actually say — and how they may help with hair concerns and scalp care.

09:14 - 05:32
luistertijd 09:14 - leestijd 05:32

Ten years ago, I heard at a conference that the scalp was far more important than we were communicating in beauty. Still, for years, haircare remained largely about shine, volume, frizz control and repair. Now the scalp has suddenly become a big thing, and there is a new term for it: skinification of hair. But what does that actually mean — and why is this attention emerging now? Could we not simply talk about the scalp?

It stayed with me, even though for a long time very little happened with it. For years I wondered why, in skin and skincare, we had become so linguistically well trained as journalists and consumers, while everything around hair and haircare remained so vague. When it came to the scalp, we barely got beyond dandruff.

Fast read

In recent years, there has been much more attention for the scalp. Knowledge has deepened: the microbiome is being studied, and researchers are looking more closely into the hair follicle. But honestly, how do we then end up suddenly talking about the skinification of hair? As if we would not understand it otherwise. I do not really want to go along with the term, but because social media has such a strong pull, I do feel called, as a journalist, to give it context.

It is not really about hair, but about the scalp

The term skinification of hair is confusing, because hair itself is not skin, even though it is made from a skin-related substance: keratin. Do not mix the two up. The outer layer of the skin, the horny layer, also contains keratin and is technically dead, but it is continuously renewed and regulated from below. A hair, once it has grown out of your scalp, is simply “finished”. It can no longer truly repair itself. Hair is dead material.

The scalp is a different story

Your scalp is living tissue. It contains sebaceous glands, blood vessels, nerves, immune cells, micro-organisms and hair follicles. It can become oily, dry, itchy, red, flaky, inflamed and sensitive to products or stress. It is not a passive piece of skin from which hair happens to grow, but a living ecosystem. Just like the skin on the rest of your body.

That is why I think it is a good thing that the scalp is finally getting this attention, and that we are moving beyond the familiar old problem of dandruff. Products are now being launched to help remove dead skin cell build-up and styling residue from the scalp. We are seeing calming tonics, formulas with hyaluronic acid, niacinamide, salicylic acid, glycolic acid, caffeine, prebiotics, postbiotics, and claims around the microbiome or a healthy scalp barrier.

Why only now?

The rise of scientific research into the microbiome, including the skin microbiome, has accelerated innovation. And in its slipstream, the all-important hair follicle has also come into sharper focus.

That tiny pouch, the womb of the hair…

Research into hair loss now looks much further than hormones alone. The hair follicle is increasingly seen as a small organ within a living skin environment, influenced by the microbiome, sebum, inflammation, oxidative stress, blood flow and possibly mechanical tension. In androgenetic alopecia, for instance, differences have been found in the microbial environment of hair follicles, although that does not yet prove that the microbiome is the cause of hair loss.

For women around and after menopause, the picture becomes even more complex. Hair can become thinner due to an interplay of age, hormonal changes, follicle sensitivity, general health, stress, nutrition, thyroid function, medication and genetic predisposition. So it is rarely as simple as: less oestrogen, therefore more hair loss.

Which claims belong to skinification?

When you make the term skinification of hair concrete, what you mostly see are scalp claims. Brands no longer say only “for shiny hair”, but also “for a healthy scalp”, “for balance”, “for comfort”, “against itching”, “for the barrier” or “for the microbiome”.

With a scalp peel containing salicylic acid or glycolic acid, you now see claims that sebum, product residue and dead skin cells are loosened, so the hair follicles are no longer clogged.

Can you hydrate hair?

Hyaluronic acid in hair products requires precision. On the scalp, it makes sense: just as on facial skin, it can bind moisture and make the skin feel more comfortable. On the hair fibre, the story is different. There, “hydration” usually does not mean adding water, but making the fibre more supple, reducing moisture loss and helping control frizz caused by humidity fluctuations. Damaged hair is more porous and reacts more strongly to humidity.

Also worth remembering: what we call dry hair is often a combination of damaged cuticles, lipid loss, increased friction and a less smooth, light-reflecting surface. A conditioner or mask makes the fibre more supple, smoother, less frizzy and less vulnerable to friction and humidity changes.

What does caffeine do?

Caffeine is also appearing more often in scalp care. The ingredient is being studied in relation to hair loss and the hair follicle, and there are studies showing favourable effects of topical caffeine — meaning applied from the outside, not through diet. But that does not mean every caffeine shampoo solves hair loss.

For you as a consumer, what matters most is how the promise is worded. “Fuller-looking hair” or “less hair breakage” is not the same as “stimulates hair growth”. As soon as a brand uses big words about hair growth or stopping hair loss, you are allowed to become critical: those claims require solid evidence, and often medical nuance too.

How do you support the microbiome?

Microbiome claims in scalp care require nuance. In scalp care, it is usually not about live probiotics, as in some specialised skincare, but about mild cleansing, barrier-friendly formulas, prebiotics, postbiotics or ferments.

Claims such as microbiome-friendly or supports the scalp microbiome make sense. But if a brand says it truly rebalances the scalp flora, there needs to be specific evidence for that. In haircare, for now, I mostly see supportive microbiome language.

What is actually measured?

Sebum measurements can show whether a product influences scalp oiliness. Redness measurements can say something about irritation or soothing. pH measurements may be relevant for a sensitive scalp or after cleansing. TEWL measurements — transepidermal water loss — give an impression of barrier function: the more water the skin loses, the more vulnerable or disrupted the barrier may be.

These are concepts from the skincare world, but they are also logical on the scalp. However, measuring them there is more challenging, because the hair is in the way. Measuring on a cheek is easier than measuring between hairs. That is why measurement methods need to be adapted, with smaller probes, image analysis or measurements close to the hairline. Trichoscopy is a particularly useful method here.

Trichoscopy

The hair follicle is increasingly seen as a small organ within a living skin environment, where sebum, microbiome, inflammation, oxidative stress, blood flow and possibly tissue tension all play a role.

With trichoscopy — a small camera that allows doctors or specialised skin therapists to view the scalp and hair roots magnified on a screen — you can see whether there is thinning hair, miniaturisation, redness, flaking, excess sebum or empty follicular openings.

And then the next step becomes more targeted. If there is a lot of sebum and flaking, you may think first of better cleansing, exfoliating scalp care or an anti-dandruff approach. If there is redness and sensitivity, calming and barrier-friendly formulas make more sense. If hair loss persists, medical assessment, hormonal checks, blood tests or evidence-based treatments such as minoxidil come into view more quickly. If the scalp feels tight or tense, massage, relaxation or attention to blood flow may be useful, although that is less strongly substantiated.

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